John Haughey

On 22/09/2015 I commenced an investigation into the death of John HAUGHEY. The investigation concluded at the end of the inquest 10th February 2017. The conclusion of the inquest was John HAUGHEY was admitted to Hull Royal Infirmary with a nine month history of increasing confusion. Whilst on Ward 80, on the 6th September 2015 he consumed a large amount of a hand wash that contained alcohol in a high concentration and developed acute alcohol toxicity which was treated by elective ventilation in the expectation that the alcohol would be naturally metabolised. His airway was unprotected for a period of seven hours between the 6th and 7th September 2015 and as a result it is likely he developed an aspiration pneumonitis which progressed to a bacterial bronchopneumonia affecting both lungs from which he died at Hull Royal Infirmary, Anlaby Road, Hull on the 12th September 2015. The medical cause of death being:

1a) Bronchopneumonia 1b) Acute alcohol toxicity

2) Acute delirium; coronary artery atherosclerosis.

John Haughey had a nine month history of increasing confusion prior to his admission to Hull Royal Infirmary in September 2015. He had a past medical history of agitation and depression and had received treatment with anti-depressant drugs. On the basis of radio diagnosis, it seems likely that he may have been developing vascular dementia by the demonstration on a brain scan of small vessel disease. Mr Haughey was admitted to Ward 80 at Hull Royal Infirmary and was noted to be agitated, combative and confused. On the 6th September 2015, he drank an unknown quantity of an alcohol-based hand sanitiser which was in a dispenser at the foot of his bed. The proprietary name of the sanitiser was “Purell Advanced Hygienic Hand Sanitising Foam”. It contains ethyl alcohol at a concentration of approximately 75%. ##DW<<corAddress>> Tel ##DW<<corTel>> | Fax ##DW<<corFax>>

Advice was given by the Tox-Based Service about the composition of the hand sanitiser and it was suggested that he should be observed clinically for depression of his level of consciousness and respiration as well as for the development of a metabolic acidosis. He was initially aggressive and combative and was prescribed Lorazepam and Haloperidol.

There was an inadequate frequency of observations instituted to record his vital signs, as it would have been predictable in the worst case scenario that he had ingested a considerable amount of alcohol and would have lapsed into coma within two hours. When first measured, his blood alcohol level was 463 milligrams per 100 millilitres of blood at 0153 hours on the 7th September 2015, this fell to 354 milligrams per 100 millilitres by 1157 hours on the same day.

He was treated in the intensive care unit and a plan to allow the alcohol to be naturally metabolised was formulated, during this time airway and ventilatory support was given. Following removal of the endotracheal tube in the Intensive Care Unit Mr Haughey’s airways were noted to contain foul smelling sputum. He required further sedative drugs following extubation to address his agitation and confusion. On the 11th September 2015 he suffered an episode of aspiration of food and as a result a serious deterioration in his condition occurred. A chest x-ray was taken which showed evidence of pneumonic changes which, on balance, were pre-existing and not caused by the episode of aspiration of ice cream. Mr Haughey’s condition progressively deteriorated and he died at 2052 hours on the 12th September 2015.